| CASE REPORT |
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| Year : 2017 | Volume
: 58
| Issue : 1 | Page : 50-52 |
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Persistent hypocalcemia and hungry bone syndrome after parathyroidectomy and renal transplantation in a patient with end-stage renal disease
Hamid Tayyebi-Khosroshahi1, Farahnoush Farnood1, Maryam Ghorbanian2, Farid Karkon-Shayan3, Mohammad Naghavi-Behzad4
1 Chronic Renal Disease Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
2 Liver and Gastrointestinal Diseases Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
3 Students' Research Committee, Tabriz University of Medical Sciences, Tabriz, Iran
4 Students' Research Committee, Tabriz University of Medical Sciences; Medical Philosophy and History Research Center, Tabriz University of Medical Sciences, Tabriz, Iran
Correspondence Address:
Mohammad Naghavi-Behzad
Medical Philosophy and History Research Center, Tabriz University of Medical Sciences, Daneshgah Street, Tabriz, Eastern Azerbaijan
Iran
 Source of Support: None, Conflict of Interest: None  | Check |
DOI: 10.4103/0300-1652.218416
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Hungry bone syndrome (HBS) defines as persistent and severe hypocalcemia after parathyroidectomy surgery. It is treated by oral or venous discrimination of calcium carbonate. The present treatment is mostly effective. Hereby, we describe a 60-year-old man who had developed hyperparathyroidism secondary to end-stage renal disease and then parathyroidectomy was performed for him twice before renal transplantation. Up to 500 vials of calcium gluconate (100 mg/ml calcium gluconate 10%) were administered for him to control serum calcium level after parathyroidectomy and renal transplantation. Furthermore, high-dose calcium carbonate was administered for his outpatient care. Therefore, HBS, which was resistant to standard treatment, was detected for him.
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